| Pain and Neuroimmune Interactions 2000 Edition Contributor(s): Saadé, Nayef E. (Editor), Jabbur, Suhayl J. (Editor), Apkarian, A. Vania (Editor) |
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ISBN: 0306463717 ISBN-13: 9780306463716 Publisher: Springer OUR PRICE: $104.49 Product Type: Hardcover - Other Formats Published: April 2000 Annotation: This volume is a result of an International Symposium on pain and neuroimmune interactions, held in Beirut, Lebanon, in May of 1999. The results of research on the neural mechanisms that relate tissue damage to pain show that the sensation of pain and suffering can be considered as part of mechanisms that involve not only sizeable areas in the brain but also simultaneous activations of the immune and the endocrine systems. Pain involves the sharing of molecular mechanisms between the nervous, immune and endocrine systems that can interact at peripheral and, ultimately, central levels. Chronic pain can then be viewed as a corollary of the imbalance in the cross-talk between these systems, which could lead to new treatment strategies. The aim of this volume is not to deal with acute pain that serves as an alarm signal, but to attempt to explain the molecular mechanisms of chronic pain considered as a multifactorial syndrome or disease. |
| Additional Information |
| BISAC Categories: - Medical | Neurology - Medical | Pain Medicine - Medical | Immunology |
| Dewey: 616.047 |
| LCCN: 00022562 |
| Physical Information: 0.63" H x 7" W x 10" (1.47 lbs) 244 pages |
| Descriptions, Reviews, Etc. |
| Publisher Description: For those of us involved in research on the neural mechanisms that relate tissue damage to pain. it is becoming more evident that the sensation of pain and suffering could be considered as part of a mechanism that involves not only sizeable areas in the brain but also simultaneous activations of the immune and the endocrine systems as well. A consensus is growing among specialists in the field that pain involves the sharing of molecular mechanisms between the nervous, immune and endocrine systems that can interact at peripheral and, ultimately, central levels. Furthermore, chronic pain could then be looked upon as a corollary of the imbalance in the cross talk between these systems, which could lead to new treatment strategies. The aim of this book is not to deal with acute pain that serves as an alarm signal, but to attempt to explain the molecular mechanisms of chronic pains considered as a multifactorial syndrome or disease. |
